![]() The conference is five days long and held in a remote location to increase the sense of camaraderie and create scientific communities, with lasting collaborations and friendships. The conference program includes a diverse range of speakers and discussion leaders from institutions and organizations worldwide, concentrating on the latest developments in the field. Some research is investigating drugs that promote myelin repair, which would mean that damage could be reversed and function improved.The Dendrites: Molecules, Structure and Function GRC is a premier, international scientific conference focused on advancing the frontiers of science through the presentation of cutting-edge and unpublished research, prioritizing time for discussion after each talk and fostering informal interactions among scientists of all career stages. Some research is looking into drugs that protect nerves from damage and so halt or slow down the progression of MS. ![]() Remyelination and neuroprotection are potential areas where new treatments could be developed. Messages to or from that part of the central nervous system are permanently blocked, resulting in symptoms that do not improve for you. Should the area of damage become too large, this rerouting process is no longer able to compensate. Messages may take longer to get through but your symptoms will improve to some extent. This ability to adapt to avoid damaged areas is called plasticity. Your central nervous system is able to overcome small areas of nerve damage by rerouting messages using undamaged nerve cells. If an axon is left without the protection of myelin it will be more vulnerable to damage and may die. Eventually, they may not be able to produce more myelin. Remyelination tends to occur in the earlier stages of MS but, with repeated relapses or attacks, oligodendrocytes become damaged and destroyed. Although the new myelin can work effectively, it tends to be thinner than unaffected myelin and so messages through the affected nerves may not be as fast as before the attack. Once the inflammation caused by the immune attack is over, it is possible for the body to replace damaged myelin. They include oligodendrocytes which produce myelin. Nerve cells are surrounded by support cells called glial cells. The thickness of the myelin sheath and the size of the gap between nodes determine the speed of messages, which can be as fast as 120 metres/second (268mph). Nerve messages leap along the axon from node to node. The myelin sheath has short gaps about one micrometre apart known as Nodes of Ranvier. Myelin acts as insulation to the axon and prevents messages becoming interrupted. The axon is surrounded by a sheath of fatty protein called myelin. They are collected by receptors on the dendrites of neighbouring neurons, and the message continues on its way. To cross the synapse, neurotransmitters are released at the end of the neuron. The synapse is the space between an axon and a dendrite of another neuron. Information enters the neuron via the dendrites, passes through the cell body and then along the axon until it reaches the synapse. The axon can be as long as one metre, making neurons some of the longest cells in the body. A neuron usually has a number of dendrites but only one axon, although this axon may have extensive branching. Neurons have specialised extensions called dendrites and axons. Pharmaceutical and other industry supporters.
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